There are no files associated with this item.
Full metadata record
DC Field | Value | Language |
---|---|---|
dc.citation.endPage | 471 | - |
dc.citation.number | 5 | - |
dc.citation.startPage | 463 | - |
dc.citation.title | MOLECULES AND CELLS | - |
dc.citation.volume | 34 | - |
dc.contributor.author | Park, Chan Young | - |
dc.contributor.author | Chun, Hyun Jin | - |
dc.contributor.author | Park, Hyeong Cheol | - |
dc.contributor.author | Koo, Sung Cheol | - |
dc.contributor.author | Lee, Ju Huck | - |
dc.contributor.author | Choi, Man Soo | - |
dc.contributor.author | Kang, Chang Ho | - |
dc.contributor.author | Baek, Dongwon | - |
dc.contributor.author | Cheong, Yong Hwa | - |
dc.contributor.author | Yun, Dae-Jin | - |
dc.contributor.author | Chung, Woo Sik | - |
dc.contributor.author | Cho, Moo Je | - |
dc.contributor.author | Kim, Min Chul | - |
dc.date.accessioned | 2023-12-22T04:37:52Z | - |
dc.date.available | 2023-12-22T04:37:52Z | - |
dc.date.created | 2015-01-08 | - |
dc.date.issued | 2012-11 | - |
dc.description.abstract | Nitric oxide (NO) is known for its role in the activation of plant defense responses. To examine the involvement and mode of action of NO in plant defense responses, we introduced calmodulin-dependent mammalian neuronal nitric oxide synthase (nNOS), which controls the CaMV35S promoter, into wild-type and NahG tobacco plants. Constitutive expression of nNOS led to NO production and triggered spontaneous induction of leaf lesions. Transgenic plants accumulated high amounts of H2O2, with catalase activity lower than that in the wild type. nNOS transgenic plants contained high levels of salicylic acid (SA), and they induced an array of SA-, jasmonic acid (JA)-, and/or ethylene (ET)-related genes. Consequently, NahG co-expression blocked the induction of systemic acquired resistance (SAR)-associated genes in transgenic plants, implying SA is involved in NO-mediated induction of SAR genes. The transgenic plants exhibited enhanced resistance to a spectrum of pathogens, including bacteria, fungi, and viruses. Our results suggest a highly ranked regulatory role for NO in SA-, JA-, and/or ET-dependent pathways that lead to disease resistance. | - |
dc.identifier.bibliographicCitation | MOLECULES AND CELLS, v.34, no.5, pp.463 - 471 | - |
dc.identifier.doi | 10.1007/s10059-012-0213-0 | - |
dc.identifier.issn | 1016-8478 | - |
dc.identifier.scopusid | 2-s2.0-84870380415 | - |
dc.identifier.uri | https://scholarworks.unist.ac.kr/handle/201301/9999 | - |
dc.identifier.url | http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84870380415 | - |
dc.identifier.wosid | 000311516400007 | - |
dc.language | 영어 | - |
dc.publisher | KOREAN SOC MOLECULAR & CELLULAR BIOLOGY | - |
dc.title | Constitutive expression of mammalian nitric oxide synthase in tobacco plants triggers disease resistance to pathogens | - |
dc.type | Article | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
Items in Repository are protected by copyright, with all rights reserved, unless otherwise indicated.
Tel : 052-217-1404 / Email : scholarworks@unist.ac.kr
Copyright (c) 2023 by UNIST LIBRARY. All rights reserved.
ScholarWorks@UNIST was established as an OAK Project for the National Library of Korea.