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Ko, Myunggon
Cancer Epigenetics Lab.
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dc.citation.endPage 4503 -
dc.citation.number 17 -
dc.citation.startPage 4501 -
dc.citation.title BLOOD -
dc.citation.volume 118 -
dc.contributor.author Ko, Myunggon -
dc.contributor.author Rao, Anjana -
dc.date.accessioned 2023-12-22T05:43:22Z -
dc.date.available 2023-12-22T05:43:22Z -
dc.date.created 2015-07-24 -
dc.date.issued 2011-10 -
dc.description.abstract The TET-family enzymes TET1, TET2, and TET3 influence DNA methylation by modifying 5-methylcytosine. Somatic loss-of-function mutations in TET2 are frequently observed in myeloid neoplasms. In this issue of Blood, Li et al. demonstrate that ablation of Tet2 alters the homeostasis and function of hematopoietic stem cells (HSCs) and induces leukemia in mice, as also reported by other groups. -
dc.identifier.bibliographicCitation BLOOD, v.118, no.17, pp.4501 - 4503 -
dc.identifier.doi 10.1182/blood-2011-08-373357 -
dc.identifier.issn 0006-4971 -
dc.identifier.scopusid 2-s2.0-80055088609 -
dc.identifier.uri https://scholarworks.unist.ac.kr/handle/201301/12565 -
dc.identifier.url http://www.bloodjournal.org/content/118/17/4501?sso-checked=true -
dc.identifier.wosid 000296368700004 -
dc.language 영어 -
dc.publisher NW SUITE 200 -
dc.title TET2: epigenetic safeguard for HSC -
dc.type Article -
dc.description.journalRegisteredClass scie -
dc.description.journalRegisteredClass scopus -

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