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Suh, Pann-Ghill
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dc.citation.endPage 1655 -
dc.citation.number 8 -
dc.citation.startPage 1645 -
dc.citation.title JOURNAL OF CELLULAR PHYSIOLOGY -
dc.citation.volume 231 -
dc.contributor.author Poli, Alessandro -
dc.contributor.author Billi, Anna Maria -
dc.contributor.author Mongiorgi, Sara -
dc.contributor.author Ratti, Stefano -
dc.contributor.author McCubrey, James A. -
dc.contributor.author Suh, Pann-Ghill -
dc.contributor.author Cocco, Lucio -
dc.contributor.author Ramazzotti, Giulia -
dc.date.accessioned 2023-12-21T23:36:29Z -
dc.date.available 2023-12-21T23:36:29Z -
dc.date.created 2016-05-30 -
dc.date.issued 2016-08 -
dc.description.abstract Phosphatidylinositol (PI) metabolism represents the core of a network of signaling pathways which modulate many cellular functions including cell proliferation, cell differentiation, apoptosis, and membrane trafficking. An array of kinases, phosphatases, and lipases acts on PI creating an important number of second messengers involved in different cellular processes. Although, commonly, PI signaling was described to take place at the plasma membrane, many evidences indicated the existence of a PI cycle residing in the nuclear compartment of eukaryotic cells. The discovery of this mechanism shed new light on many nuclear functions, such as gene transcription, DNA modifications, and RNA expression. As these two PI cycles take place independently of one another, understanding how nuclear lipid signaling functions and modulates nuclear output is fundamental in the study of many cellular processes. -
dc.identifier.bibliographicCitation JOURNAL OF CELLULAR PHYSIOLOGY, v.231, no.8, pp.1645 - 1655 -
dc.identifier.doi 10.1002/jcp.25273 -
dc.identifier.issn 0021-9541 -
dc.identifier.scopusid 2-s2.0-84953923806 -
dc.identifier.uri https://scholarworks.unist.ac.kr/handle/201301/19288 -
dc.identifier.url http://onlinelibrary.wiley.com/doi/10.1002/jcp.25273/abstract;jsessionid=A113146E8AB27D135F703C88753A2F18.f01t03 -
dc.identifier.wosid 000375123600003 -
dc.language 영어 -
dc.publisher WILEY-BLACKWELL -
dc.title Nuclear Phosphatidylinositol Signaling: Focus on Phosphatidylinositol Phosphate Kinases and Phospholipases C -
dc.type Article -
dc.description.isOpenAccess FALSE -
dc.relation.journalWebOfScienceCategory Cell Biology; Physiology -
dc.relation.journalResearchArea Cell Biology; Physiology -
dc.description.journalRegisteredClass scie -
dc.description.journalRegisteredClass scopus -
dc.subject.keywordPlus RISK MYELODYSPLASTIC SYNDROMES -
dc.subject.keywordPlus CELL-CYCLE PROGRESSION -
dc.subject.keywordPlus ACUTE MYELOID-LEUKEMIA -
dc.subject.keywordPlus GROWTH-FACTOR-I -
dc.subject.keywordPlus MURINE ERYTHROLEUKEMIA-CELLS -
dc.subject.keywordPlus SWISS 3T3-CELLS -
dc.subject.keywordPlus MESSENGER-RNA -
dc.subject.keywordPlus MYOGENIC DIFFERENTIATION -
dc.subject.keywordPlus ERYTHROID-DIFFERENTIATION -
dc.subject.keywordPlus 4-PHOSPHATE 5-KINASE -

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